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NEET MDS Synopsis

TEMPOROMANDIBULAR JOINT
Dental Anatomy

TEMPOROMANDIBULAR JOINT

There are three kind of joints:
 

·  Fibrous
Two bones connected with fibrous tissue
Examples
suture (little or no movement)
gomphosis (tooth - PDL - bone)
syndesmosis (fibula & tibia, radius and ulna; interosseous ligament)

·  Cartilagenous
Two subtypes:
2a) primary: bone<--->cartilage (costochondral joint)
2b) secondary: bone<-->cartilage<-->FT<-->cartilage<--> bone (pubic symphysis)

·  Synovial
Two bones; each articular surface covered with hyaline cartilage in most cases
The bones are united with a capsule (joint cavity)
In the capsule there is presence of synovial fluid
The capsule is lined by a synovial membrane
In many synovial joints there maybe an articular disk
Synovial joints are characterized by the presence of ligaments
Synovial joints are classified according to the number of axes of bone movement: uniaxial, biaxial, multiaxial

the shapes of articulating surfaces: planar, ginglymoid (=hinged), pivot, condyloid

The movement of the joints is controlled by muscles

The temporomandibular joint is a synovial, sliding-ginglymoid joint (humans)

Embryology of the TMJ
Primary TMJ: Meckel's cartilage --> malleus & incal cartilage. It lasts for 4 months.
Secondary TMJ: Starts developing around the third month of gestation
Two blastemas (temporal and condylar); condylar grows toward the temporal (temporal appears and ossifies first)
Formation of two cavities: inferior and upper
Appearance of disk
Bones: glenoid fossa (temporal bone) and condyle (mandible)
 

Methods of general anesthesia
Pharmacology

Methods of general anesthesia

CIRCLE SYSTEM

*HIGH-FLOW

FRESH GAS FLOW > 3 l/min.

*LOW-FLOW

FGF ok. 1l/min.

*MINIMAL-FLOW

FGF ok. 0,5 l/min.

Drugs Used in Diabetes -Insulin
Pharmacology

Insulin
Insulin is only given parenterally (subcutaneous or IV) Various preparations have different durations of action 
 





Preparation


Onset (hrs)


Peak (hrs)


Duration (hrs)



Lispro (rapid-acting)
15 min
0.5-1.5
3-4


Regular (short-acting)
0.5-1
2-4
5-7


NPH (intermediate)
1-2
6-12
18-24


Glargine (long-acting)
1
None
>24




 

Mechanism

bind transmembrane insulin receptor
activate tyrosine kinase
phosphorylate specific substrates in each tissue type
liver
↑ glycogenesis
store glucose as glycogen
muscle
↑ glycogen and protein synthesis
↑ K+ uptake 
fat
increase triglyceride storage

Clinical use

type I DM
type II DM
life-threatening hyperkalemia
increases intracellular K+
stress-induced hyperglycemia
 

Toxicity
hypoglycemia
hypersensitivity reaction (very rare)

Insulin Synthesis
first generated as preproinsulin with an A chain and B chain connected by a C peptide. 
c-peptide is cleaved from proinsulin after packaging into vesicles leaving behind the A and B chains


Glycogen Storage Diseases

Biochemistry


Glycogen Storage Diseases are genetic enzyme deficiencies associated with excessive glycogen accumulation within cells.


When an enzyme defect affects mainly glycogen storage in liver, a common symptom is hypoglycemia (low blood glucose), relating to impaired mobilization of glucose for release to the blood during fasting.
When the defect is in muscle tissue, weakness and difficulty with exercise result from inability to increase glucose entry into Glycolysis during exercise.


Various type of Glycogen storage disease are





Type


Name


Enzyme Deficient




I


Von Geirke’s Disease


Glucose -6-phosphate




II


Pompe’s Disease


(1, 4)glucosidase




III


Cori’s Disease


Debranching Enzymes




IV


Andersen’s Disease


Branching Enzymes




V


McArdle’s Disease


Muscles Glycogen Phosphorylase





OXYMETAZOLINE
Pharmacology

OXYMETAZOLINE
 

It is a directly acting sympathomimetic amine used in symptomatic relief in nasal congestion which increases mucosal secretion.

It is used:
- As a nasal decongestant in allergic rhinitis, with or without the addition of antazoline or sodium chromoglycate. 
- As an ocular decongestant in allergic conjunctivitis.

Compounds like naphazoline and xylometazoline are relatively selective α2 agonists, which on topical application produce local vasoconstriction.

Relationship Classification
Dental Anatomy

Angle classified these relationships by using the first permanent molars

Normal or neutral occlusion (ideal):

Mesiobuccalgroove of the mandibular first molar align with the mesiobuccal cusp of the max laxy first permanent molar

ClassI  malocclusion  normal molar relationships with alterations to other characteristics of the occlusion such as versions, crossbites, excessive overjets, or overbites

 

Class II malocclusion a distal relation of the mesiobuccal groove of the mandibular first permanent molar to the mesiobuccal cusp of the maxillary first permanent molar

 

Division I: protruded maxillary anterior teeth

Division II: one or more maxillary anterior teeth retruded

Class III  malocclusion a mesial relation of the mesiobuccal groove of the mandibular first permanent molar to the mesiobuccal cusp of the maxillary molar

Impression Materials -Classification
Dental Materials

Classification

Rigid impression materials

(1) Plaster
(2) Compound
(3) Zinc oxide-eugenol

Flexible hydrocolloid impression materials

(I) Agar-agar (reversible hydrocolloid)
(2) Alginate (irreversible hydrocolloid)

Flexible, elastomeric, or rubber impression materials

(1) Polysulfide rubber (mercaptan rubber)
(2) Silicone rubber (condensation silicone)
(3) Polyether rubber
(4) Polyvinyl siloxane (addition silicone)
 

The Optic Nerve
Anatomy


This is the second cranial nerve (CN II) and is the nerve of sight.

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