NEET MDS Shorts
93349
Periodontics
76870
General Medicine
In liver disease, especially cirrhosis, there is a decrease in hepatic synthesis of clotting factors, particularly factor II (prothrombin). Vitamin K deficiency is also common in liver disease, which further contributes to hypoprothrombinemia. The deficiency of fibrinogen (factor I) would also result in a bleeding diathesis but is less common in liver disease. Factors V and VIII are also synthesized in the liver but are less frequently affected than factor II in such cases.
84128
Oral PathologyExposure for children should be reduced by 50% compared to adults.
96049
General Medicine
Anti-Smith (anti-Sm) antibodies are highly specific for SLE and are found in about 20-30% of patients. While antinuclear antibodies and anti-double stranded DNA antibodies are also associated with SLE, they are less specific as they can also be present in other conditions.
72251
Oral SurgeryLatreal Pterygoid is inserted on Mandible
53287
Oral Medicine
Chronic venous insufficiency is characterized by a range of symptoms and physical findings, including varicose veins, pitting edema, and venous ulcers, particularly around the medial malleolus (ankle). These symptoms result from impaired venous return and increased venous pressure in the lower extremities.
60545
EndodonticsEndodontic failure in maxillary first molars is often attributed to the complex canal morphology, particularly the mesiobuccal root. The inability to identify and appropriately treat all existing canals, specifically the second mesiobuccal (MB2) canal or "fourth canal," is considered a primary reason for root canal treatment failure
40194
INI CET
The occlusal plane must be leveled first, usually by crowning or selective grinding of the extruded tooth.
99358
Physiology
The rubrospinal tract is a lateral pathway that originates in the red nucleus and is involved in the control of distal muscle movement, particularly in the control of fine motor movements of the limbs.
99977
INI CET
N-acetylcysteine (NAC) is the antidote for paracetamol toxicity. It works by replenishing glutathione stores in the liver, which neutralizes the toxic metabolite NAPQI.