NEET MDS Lessons
Dental Anatomy
Development of occlusion.
A. Occlusion usually means the contact relationship in function. Concepts of occlusion vary with almost every specialty of dentistry.
Centric occlusion is the maximum contact and/or intercuspation of the teeth.
B. Occlusion is the sum total of many factors.
1. Genetic factors.
-Teeth can vary in size. Examples are microdontia (very small teeth) and macrodontia (very large teeth). Incidentally, Australian aborigines have the largest molar tooth size—some 35% larger than the smallest molar tooth group
-The shape of individual teeth can vary (such as third molars and the upper lateral incisors.)
-They can vary when and where they erupt, or they may not erupt at all (impaction).
-Teeth can be congenitally missing (partial or complete anodontia), or there can be extra (supernumerary) teeth.
-The skeletal support (maxilla/mandible) and how they are related to each other can vary considerably from the norm.
2. Environmental factors.
-Habits can have an affect: wear, thumbsucking, pipestem or cigarette holder usage, orthodontic appliances, orthodontic retainers have an influence on the occlusion.
3.Muscular pressure.
-Once the teeth erupt into the oral cavity, the position of teeth is affected by other teeth, both in the same dental arch and by teeth in the opposing dental arch.
-Teeth are affected by muscular pressure on the facial side (by cheeks/lips) and on the lingual side (by the tongue).
C. Occlusion constantly changes with development, maturity, and aging.
1 . There is change with the eruption and shedding of teeth as the successional changes from deciduous to permanent dentitions take place.
2. Tooth wear is significant over a lifetime. Abrasion, the wearing away of the occlusal surface reduces crown height and alters occlusal anatomy.
Attrition of the proximal surfaces reduces the mesial-distal dimensions of the teeth and significantly reduces arch length over a lifetime.
Abraision is the wear of teeth by agencies other than the friction of one tooth against another.
Attrition is the wear of teeth by one tooth rubbing against another
3. Tooth loss leaves one or more teeth without an antagonist. Also, teeth drift, tip, and rotate when other teeth in the arch are extracted.
Tooth development is commonly divided into the following stages: the bud stage, the cap, the bell, and finally maturation. The staging of tooth development is an attempt to categorize changes that take place along a continuum; frequently it is difficult to decide what stage should be assigned to a particular developing tooth. This determination is further complicated by the varying appearance of different histological sections of the same developing tooth, which can appear to be different stages.
Bud stage
The bud stage is characterized by the appearance of a tooth bud without a clear arrangement of cells. The stage technically begins once epithelial cells proliferate into the ectomesenchyme of the jaw. The tooth bud itself is the group of cells at the end of the dental lamina.
Amelogenesis and Enamel
Enamel is highly mineralized: 85% hydroxyapatite crystals
Enamel formation is a two-step process
The first step produces partially mineralized enamel: 30% (secretory)
The second step: Influx of minerals, removal of water and organic matrix (maturative)
Again, dentin is the prerequisite of enamel formation (reciprocal induction)
Stratum intermedium: high alkaline phosphatase activity
Differentiation of ameloblasts: Increase in glycogen contents
Formation of the enamel matrix
Enamel proteins, enzymes, metalloproteinases, phosphatases, etc.
Enamel proteins: amelogenins (90%), enamelin, tuftelin, and amelin
Amelogenins: bulk of organic matrix
Tuftelin: secreted at the early stages of amelogenesis (area of the DE junction)
Enamelin: binds to mineral
Amelin
Mineralization of enamel
No matrix vesicles
Immediate formation of crystallites
Intermingling of enamel crystallites with dentin
"Soft" enamel is formed
Histologic changes
Differentiation of inner enamel epithelium cells. They become ameloblasts
Tomes' processes: saw-toothed appearance
Collapse of dental organ
Formation of the reduced enamel epithelium
Hard tissue formation (Amelogenesis )
Enamel formation is called amelogenesis and occurs in the crown stage of tooth development. "Reciprocal induction" governs the relationship between the formation of dentin and enamel; dentin formation must always occur before enamel formation. Generally, enamel formation occurs in two stages: the secretory and maturation stages. Proteins and an organic matrix form a partially mineralized enamel in the secretory stage; the maturation stage completes enamel mineralization.
In the secretory stage, ameloblasts release enamel proteins that contribute to the enamel matrix, which is then partially mineralized by the enzyme alkaline phosphatase. The appearance of this mineralized tissue, which occurs usually around the third or fourth month of pregnancy, marks the first appearance of enamel in the body. Ameloblasts deposit enamel at the location of what become cusps of teeth alongside dentin. Enamel formation then continues outward, away from the center of the tooth.
In the maturation stage, the ameloblasts transport some of the substances used in enamel formation out of the enamel. Thus, the function of ameloblasts changes from enamel production, as occurs in the secretory stage, to transportation of substances. Most of the materials transported by ameloblasts in this stage are proteins used to complete mineralization. The important proteins involved are amelogenins, ameloblastins, enamelins, and tuftelins. By the end of this stage, the enamel has completed its mineralization.
Mandibular Second Deciduous Molar.
-This tooth resembles the lower first permanent molar that is d
istal to it in the dental arch.
-There are two roots and five cusps. The three buccal cusps are all about the same size. This is in contrast to the lower first molar where the 'distal' cusp is smaller that the mesiobuccal and distobuccal cusps.
-The distal of the three buccal cusps may be shifted of onto the distal marginal ridge.
NOTE
-Upper molars have three roots, lowers have two roots.
-Upper and lower second deciduous molars resemble first permanent molars in the same quadrant.
-Upper first deciduous molars vaguely resemble upper premolars. -Lower first deciduous molars are odd and unique unto themselves.
-First deciduous molars (upper and lower) have a prominent bulge of enamel on the buccal at the mesial. These help in determining right and left.
Enamel
Structural characteristics and microscopic features
a. Enamel rods or prisms
(1) Basic structural unit of enamel.
(2) Consists of tightly packed hydroxyapatite crystals. Hydroxyapatite crystals in enamel are four times larger and more tightly packed than hydroxyapatite found in other calcified
tissues (i.e., it is harder than bone).
(3) Each rod extends the entire thickness of enamel and is perpendicular to the dentinoenamel junction (DEJ).
b. Aprismatic enamel
(1) The thin outer layer of enamel found on the surface of newly erupted teeth.
(2) Consists of enamel crystals that are aligned perpendicular to the surface.
(3) It is aprismatic (i.e., prismless) and is more mineralized than the enamel beneath it.
(4) It results from the absence of Tomes processes on the ameloblasts during the final stages of enamel deposition.
c. Lines of Retzius (enamel striae)
(1) Microscopic features
(a) In longitudinal sections, they are observed as brown lines that extend from the DEJ to the
tooth surface.
(b) In transverse sections, they appear as dark, concentric rings similar to growth rings in a tree.
(2) The lines appear weekly during the formation of enamel.
(3) Although the cause of striae formation is unknown, the lines may represent appositional or incremental growth of enamel. They may also result from metabolic disturbances of ameloblasts.
(4) Neonatal line
(a) An accentuated, dark line of Retzius that results from the effect of physiological changes
on ameloblasts at birth.
(b) Found in all primary teeth and some cusps of permanent first molars.
d. Perikymata
(1) Lines of Retzius terminate on the tooth surface in shallow grooves known a perikymata.
(2) These grooves are usually lost through wear but may be observed on the surfaces of developing teeth or nonmasticatory surfaces of formed teeth.
e. Hunter-Schreger bands
(1) Enamel rods run in different directions. In longitudinal sections, these changes in direction result in a banding pattern known as HunterSchreger bands.
(2) These bands represent an optical phenomenon of enamel and consist of a series of alternating dark and light lines when the section is viewed with reflected or polarized
light.
f. Enamel tufts
(1) Consist of hypomineralized groups of enamel rods.
(2) They are observed as short, dark projections found near or at the DEJ.
(3) They have no known clinical significance.
g. Enamel lamellae
(1) Small, sheet-like cracks found on the surface of enamel that extend its entire thickness.
(2) Consist of hypocalcified enamel.
(3) The open crack may be filled with organic material from leftover enamel organ components, connective tissues of the developing tooth, or debris from the oral cavity.
(4) Both enamel tufts and lamellae may be likened to geological faults in mature enamel.
h. Enamel spindle
(1) Remnants of odontoblastic processes that become trapped after crossing the DEJ during the differentiation of ameloblasts.
(2) Spindles are more pronounced beneath the cusps or incisal edges of teeth (i.e., areas where occlusal stresses are the greatest).
The very first histological evidence of tooth development appear during the second month of intrauterine life. Calcification of deciduous incisors begins at 3-4 months in utero.
INNERVATION OF THE DENTIN-PULP COMPLEX
- Dentine Pulp
- Dentin
- Nerve Fibre Bundle
- Nerve fibres
The nerve bundles entering the tooth pulp consist principally of sensory afferent fibers from the trigeminal nerve and sympathetic branches from the superior cervical ganglion. There are non-myelinated (C fibers) and myelinated (less than non, A-delta, A-beta) fibers. Some nerve endings terminate on or in association with the odontoblasts and others in the predentinal tubules of the crown. Few fibers are found among odontoblasts of the root.
In the cell-free zone one can find the plexus of Raschkow.