NEET MDS Lessons
General Medicine
Naber’s Probe and Furcation Involvement
Furcation involvement is a critical aspect of periodontal disease that affects the prognosis of teeth with multiple roots. Naber’s probe is a specialized instrument designed to assess furcation areas, allowing clinicians to determine the extent of periodontal attachment loss and the condition of the furcation. This lecture will cover the use of Naber’s probe, the classification of furcation involvement, and the clinical significance of these classifications.
Naber’s Probe
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Description: Naber’s probe is a curved, blunt-ended instrument specifically designed for probing furcation areas. Its unique shape allows for horizontal probing, which is essential for accurately assessing the anatomy of multi-rooted teeth.
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Usage: The probe is inserted horizontally into the furcation area to evaluate the extent of periodontal involvement. The clinician can feel the anatomical fluting between the roots, which aids in determining the classification of furcation involvement.
Classification of Furcation Involvement
Furcation involvement is classified into four main classes using Naber’s probe:
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Class I:
- Description: The furcation can be probed to a depth of 3 mm.
- Clinical Findings: The probe can feel the anatomical fluting between the roots, but it cannot engage the roof of the furcation.
- Significance: Indicates early furcation involvement with minimal attachment loss.
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Class II:
- Description: The furcation can be probed to a depth greater than 3 mm, but not through and through.
- Clinical Findings: This class represents a range between Class I and Class III, where there is partial loss of attachment but not complete penetration through the furcation.
- Significance: Indicates moderate furcation involvement that may require intervention.
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Class III:
- Description: The furcation can be completely probed through and through.
- Clinical Findings: The probe passes from one furcation to the other, indicating significant loss of periodontal support.
- Significance: Represents advanced furcation involvement, often associated with a poor prognosis for the affected tooth.
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Class III+:
- Description: The probe can go halfway across the tooth.
- Clinical Findings: Similar to Class III, but with partial obstruction or remaining tissue.
- Significance: Indicates severe furcation involvement with a significant loss of attachment.
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Class IV:
- Description: Clinically, the examiner can see through the furcation.
- Clinical Findings: There is complete loss of tissue covering the furcation, making it visible upon examination.
- Significance: Indicates the most severe form of furcation involvement, often leading to tooth mobility and extraction.
Measurement Technique
- Measurement Reference: Measurements are taken from an imaginary tangent connecting the prominences of the root surfaces of both roots. This provides a consistent reference point for assessing the depth of furcation involvement.
Clinical Significance
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Prognosis: The classification of furcation involvement is crucial for determining the prognosis of multi-rooted teeth. Higher classes of furcation involvement generally indicate a poorer prognosis and may necessitate more aggressive treatment strategies.
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Treatment Planning: Understanding the extent of furcation involvement helps clinicians develop appropriate treatment plans, which may include scaling and root planing, surgical intervention, or extraction.
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Monitoring: Regular assessment of furcation involvement using Naber’s probe can help monitor disease progression and the effectiveness of periodontal therapy.
Contraindications and Limitations
Gingivectomy
- Major Contraindication: When pocket bottom is apical to mucogingival junction
- Risk: Inadequate attached gingiva post-surgery
Curettage Protocol
- Prerequisite: Should always be preceded by scaling and root planing
- Rationale: Reduce bacterial load before surgical intervention
GTR (Guided Tissue Regeneration)
Membrane Placement Technique
- Apical Extension: 3-4mm apical to defect margin
- Lateral Extension: 2-3mm laterally beyond defect
- Objective: Exclude epithelial and connective tissue cells
Implant Considerations
Placement Guidelines
- Distance from Mental Foramen: ≥5mm to avoid nerve damage
- Primary Stability: Critical for osseointegration
Micro-movement Tolerance
- Critical Threshold: >150μm causes fibrous tissue formation instead of osseointegration
- Clinical Implication: Importance of proper implant stability
Keratinized Gingiva and Attached Gingiva
The gingiva is an essential component of the periodontal tissues, providing support and protection for the teeth. Understanding the characteristics of keratinized gingiva, particularly attached gingiva, is crucial for assessing periodontal health.
Keratinized Gingiva
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Definition:
- Keratinized gingiva refers to the gingival tissue that is covered by a layer of keratinized epithelium, providing a protective barrier against mechanical and microbial insults.
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Areas of Keratinized Gingiva:
- Attached Gingiva:
- Extends from the gingival groove to the mucogingival junction.
- Marginal Gingiva:
- The free gingival margin that surrounds the teeth.
- Hard Palate:
- The roof of the mouth, which is also covered by keratinized tissue.
- Attached Gingiva:
Attached Gingiva
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Location:
- The attached gingiva is the portion of the gingiva that is firmly bound to the underlying alveolar bone.
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Width of Attached Gingiva:
- The width of attached gingiva varies based on location and can increase with age and in cases of supraerupted teeth.
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Measurements:
- Greatest Width:
- Found in the incisor region:
- Maxilla: 3.5 mm - 4.5 mm
- Mandible: 3.3 mm - 3.9 mm
- Found in the incisor region:
- Narrowest Width:
- Found in the posterior region:
- Maxillary First Premolar: 1.9 mm
- Mandibular First Premolar: 1.8 mm
- Found in the posterior region:
- Greatest Width:
Clinical Significance
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Importance of Attached Gingiva:
- The width of attached gingiva is important for periodontal health, as it provides a buffer zone against mechanical forces and helps maintain the integrity of the periodontal attachment.
- Insufficient attached gingiva may lead to increased susceptibility to periodontal disease and gingival recession.
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Assessment:
- Regular assessment of the width of attached gingiva is essential during periodontal examinations to identify potential areas of concern and to plan appropriate treatment strategies.
Anatomy and Histology of the Periodontium
Gingiva (normal clinical appearance): no muscles, no glands; keratinized
- Color: coral pink but does vary with individuals and races due to cutaneous pigmentation
- Papillary contour: pyramidal shape with one F and one L papilla and the col filling interproximal space to the contact area (col the starting place gingivitis)
- Marginal contour: knife-edged and scalloped
- Texture: stippled (orange-peel texture); blow air to dry out and see where stippling ends to see end of gingiva
- Consistency: firm and resilient (push against it and won’t move); bound to underlying bone
- Sulcus depth: 0-3mm
- Exudate: no exudates (blood, pus, water)
Anatomic and histological structures
Gingival unit: includes periodontium above alveolar crest of bone
a. Alveolar mucosa: histology- non-keratinized, stratified, squamous epithelium, submucosa with glands, loose connective tissue with collagen and elastin, muscles. No epithelial ridges, no stratum granulosum (flattened cells below keratin layer)
b. Mucogingival junction: clinical demarcation between alveolar mucosa and attached gingiva
c. Attached gingiva: histology- keratinized, stratified, squamous epithelium with epithelial ridges (basal cell layer, prickle cell layer, granular cell layer (stratum granulosum), keratin layer); no submucosa
- Dense connective tissue: predominantly collagen, bound to periosteum of bone by Sharpey fibers
- Reticular fibers between collagen fibers and are continuous with reticulin in blood vessels
d. Free gingival groove: demarcation between attached and free gingiva; denotes base of gingival sulcus in normal gingiva; not always seen
e. Free gingival margin: area from free gingival groove to epithelial attachment (up and over ® inside)
- Oral surface: stratified, squamous epithelium with epithelial ridges
- Tooth side surface (sulcular epithelium): non-keratinized, stratified, squamous epithelium with no epithelial ridges (basal cell and prickle cell layers)
f. Gingival sulcus: space bounded by tooth surface, sulcular epithelium, and junctional epithelium; 0-3mm depth; space between epithelium and tooth
g. Dento-gingival junction: combination of epithelial and fibrous attachment
- Junctional epithelium (epithelial attachment): attachment of epithelial cells by hemi-desmosomes and sticky substances (basal lamina- 800-1200 A, DAS-acid mucopolysaccharides, hyaluronic acid, chondroitin sulfate A, C, and B), to enamel, enamel and cementum, or cementum depending on stage of passive eruption. Length ranges from 0.25-1.35mm.
- Fibrous attachment: attachment of collagen fibers (Sharpey’s fibers) into cementum just beneath epithelial attachment; ~ 1mm thick
h. Nerve fibers: myelinated and non-myelinated (for pain) in connective tissue. Both free and specialized endings for pain, touch pressure, and temperature -> proprioception. If dentures, rely on TMJ.
i.Mesh of terminal argyophilic fibers (stain silver), some extending into epithelium
ii Meissner-type corpuscles: pressure sensitive sensory nerve encased in CT
iii.Krause-type corpuscles: temperature receptors
iv. Encapsulated spindles
i. Gingival fibers:
i. Gingivodental group:
- Group I (A): from cementum to free gingival margin
- Group II (B): from cementum to attached gingiva
- Group III (C): from cementum over alveolar crest to periosteum on buccal and lingual plates
ii. Circular (ligamentum circularis): encircles tooth in free gingiva
iii. Transeptal fibers: connects cementum of adjacent teeth, runs over interdental septum of alveolar bone. Separates gingival unit from attachment apparatus.
Transeptal and Group III fibers the major defense against stuff getting into bone and ligament.
2. Attachment apparatus: periodontium below alveolar crest of bone
Periodontal ligament: Sharpey’s fibers (collagen) connecting cementum to bone (bundle bone). Few elastic and oxytalan fibers associated with blood vessels and embedded in cementum in cervical third of tooth. Components divided as follows:
i. Alveolar crest fibers: from cementum just below CEJ apical to alveolar crest of bone
ii.Horizontal fibers: just apical to alveolar crest group, run at right angles to long axis of tooth from cementum horizontally to alveolar bone proper
iii.Oblique fibers: most numerous, from cementum run coronally to alveolar bone proper
iv. Apical fibers: radiate from cementum around apex of root apically to alveolar bone proper, form socket base
v. Interradicular fibers: found only between roots of multi-rooted teeth from cementum to alveolar bone proper
vi. Intermediate plexus: fibers which splice Sharpey’s fibers from bone and cementum
vii. Epithelial Rests of Malassez: cluster and individual epithelial cells close to cementum which are remnants of Hertwig’s epithelial root sheath; potential source of periodontal cysts.
viii. Nerve fibers: myelinated and non-myelinated; abundant supply of sensory free nerve endings capable of transmitting tactile pressure and pain sensation by trigeminal pathway and elongated spindle-like nerve fiber for proprioceptive impulses
Cementum: 45-50% inorganic; 50-55% organic (enamel is 97% inorganic; dentin 70% inorganic)
i. Acellular cementum: no cementocytes; covers dentin (older) in coronal ½ to 2/3 of root, 16-60 mm thick
ii. Cellular cementum: cementocytes; covers dentin in apical ½ to 1/3 of root; also may cover acellular cementum areas in repair areas, 15-200 mm thick
iii. Precementum (cementoid): meshwork of irregularly arranged collagen in surface of cementum where formation starts
iv. Cemento-enamel junction (CEJ): 60-65% of time cementum overlaps enamel; 30% meet end-to-end; 5-10% space between
v. Cementum slower healing than bone or PDL. If expose dentinotubules ® root sensitivity.
Alveolar bone: 65% inorganic, 35% organic
i. Alveolar bone proper (cribriform plate): lamina dura on x-ray; bundle bone receive Sharpey fibers from PDL
ii. Supporting bone: cancellous, trabecular (vascularized) and F and L plates of compact bone
Blood supply to periodontium
i. Alveolar blood vessels (inferior and superior)
A) Interalveolar: actually runs through bone then exits, main supply to alveolar bone and PDL
B) Supraperiosteal: just outside bone, to gingiva and alveolar bone
C) Dental (pulpal): to pulp and periapical area
D) Terminal vessels (supracrestal): anastomose of A and B above beneath the sulcular epithelium
E) PDL gets blood from: most from branches of interalveolar blood vessels from alveolar bone marrow spaces, supraperiosteal vessels when interalveolar vessels not present, pulpal (apical) vessels, supracrestal gingival vessels
ii. Lymphatic drainage: accompany blood vessels to regional lymph nodes (esp. submaxillary group)
- Scaling in 2 appointments within 24 hours
- Tongue brushing with 1% chlorhexidine gel
- 0.2% chlorhexidine rinse
- 1% chlorhexidine pocket irrigation
Oral Hygiene Tools
Toothbrush Bristle Diameter:
- Soft: 0.007" (0.2 mm)
- Medium: 0.012" (0.3 mm)
- Hard: 0.014" (0.4 mm)
Brushing Techniques:
- Roll: Modified Stillman
- Circular: Fones
- Vertical: Leonard
- Horizontal: Scrub
Scaling Instruments
- Gracey curettes: Only outer cutting edge
- Universal curettes: Both edges cutting
- Langer curettes: Gracey shank + Universal blade
- Best grasp: Modified pen grasp (tripod effect)
- Scaling angle: 45 – 90°
Ultrasonic Scaling
- Vibration frequency: 18,000 – 50,000 cycles/sec
Stroke Types
- Exploratory: Light feeling (with probes)
- Scaling: Short powerful pull
- Root planing: Moderate to light pull
Necrotizing Ulcerative Gingivitis (NUG)
Necrotizing Ulcerative Gingivitis (NUG), also known as Vincent's disease or trench mouth, is a severe form of periodontal disease characterized by the sudden onset of symptoms and specific clinical features.
Etiology and Predisposing Factors
- Sudden Onset: NUG is characterized by a rapid onset of symptoms, often following debilitating diseases or acute respiratory infections.
- Lifestyle Factors: Changes in living habits, such as prolonged work without adequate rest, poor nutrition, tobacco use, and psychological stress, are frequently noted in patient histories .
- Smoking: Smoking has been identified as a significant predisposing factor for NUG/NDP .
- Immune Compromise: Conditions that compromise the immune system, such as poor oral hygiene, smoking, and emotional stress, are major contributors to the development of NUG .
Clinical Presentation
- Symptoms: NUG presents with:
- Punched-out, crater-like depressions at the crest of interdental papillae.
- Marginal gingival involvement, with rare extension to attached gingiva and oral mucosa.
- Grey, pseudomembranous slough covering the lesions.
- Spontaneous bleeding upon slight stimulation of the gingiva.
- Fetid odor and increased salivation.
Microbiology
- Mixed Bacterial Infection: NUG is caused by a complex
of anaerobic bacteria, often referred to as the fusospirochetal complex,
which includes:
- Treponema vincentii
- Treponema denticola
- Treponema macrodentium
- Fusobacterium nucleatum
- Prevotella intermedia
- Porphyromonas gingivalis
Treatment
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Control of Acute Phase:
- Clean the wound with an antibacterial agent.
- Irrigate the lesion with warm water and 5% vol/vol hydrogen peroxide.
- Prescribe oxygen-releasing mouthwash (e.g., hydrogen peroxide DPF, sodium perborate DPF) to be used thrice daily.
- Administer oral metronidazole for 3 to 5 days. If sensitive to metronidazole, prescribe penicillin; if sensitive to both, consider erythromycin or clindamycin.
- Use 2% chlorhexidine in select cases for a short duration.
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Management of Residual Condition:
- Remove predisposing local factors (e.g., overhangs).
- Perform supra- and subgingival scaling.
- Consider gingivoplasty to correct any residual gingival deformities.
Causes
- Volatile sulfur compounds: H₂S, methyl mercaptan, dimethyl sulfide
Detection Methods
- Gold standard: Organoleptic rating (easiest)
- Most sensitive: Gas chromatography